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  1. 2016年7月1日 · A novel lesion mimic and early senescence 2 (lmes2) mutant was identified from Jinhui10, an indica restorer line in rice (Oryza sativa L.), which was treated with ethyl methane sulfonate (EMS). Lmes2 began to develop disease-like lesions on leaves at the

    • Yadi Xing, Dan Du, Yanhua Xiao, Tianquan Zhang, Xinlong Chen, Ping Feng, XianChun Sang, Nan Wang, Gu...
    • 6
    • 2016
    • 01 July 2016
  2. 2024年8月22日 · The occurrence of lesion mimic and early senescence is affected by multiple factors, including hormones, metabolism, and ambient signals. However, the molecular mechanism underlying this process remains incompletely understood. In this study, we identified a lesion mimic and early senescence mutant, named lmes6 ( lesion mimic and ...

  3. From a rice mutant library subjected to EMS treatment, we identified a recessive mutant, rlr1 (previously designated lmes2), that developed HR-like lesions on the leaf blade with an early senescence phenotype in the absence of pathogen infection (Xing et al., 2016).

    • 4/25/2021 1:43:15 PM
  4. 2016年7月1日 · Semantic Scholar extracted view of "Fine Mapping of a New Lesion Mimic and Early Senescence 2 (lmes2) Mutant in Rice" by Y. Xing et al.

  5. OsLMS encodes a protein with two double-stranded RNA binding motifs that is responsible for the lesion mimic phenotype and early senescence in rice (Undan et al., 2012). Here, we identified a new lesion mimic and early senescence 2 (lmes2) in rice, which was located in a 66-kb region of the short arm of chromosome 10.

  6. 2021年10月18日 · A novel light intensity-dependent lesion mimic mutant with enhanced disease resistance was physiologically, biochemically, and genetically characterized, and the causative gene was fine mapped to a 1.28 Mbp interval containing 17 high-confidence genes.

  7. 2022年3月17日 · A lesion mimic and early senescence rice mutant was obtained by soaking and mutagenizing the seeds of Xinong 1B with 0.4% zhionzhon ethyl methanesulfonate for 16 h. After four generations of selfing (2016–2018), the mutant had a stable phenotype and was named lmes5 (lesion mimic and early senescence mutant 5).

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